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Robert L. Fairchild

Staff
Immunology Medicine
Cleveland Clinic Lerner Research Institute
United States of America

Professor Infectious Diseases
Biography

Epidermal application of reactive haptens such as urushiol, the reactive agent in poison ivy, and subsequent challenge results in the development of a T cell mediated inflammatory response termed allergic contact dermatitis or contact hypersensitivity (CHS).

Research Intrest

Skin disease, Skin and cardiac allograft rejection, T cell recruitment to inflammation in peripheral tissues

List of Publications
Ishii D, Schenk AD, Baba S, Fairchild RL (2010) Role of TNFa in early chemokine production and leukocyte infiltration into heart allografts. Am. J. Transplant. 10:59-68,
Setoguchi K, Schenk AD, Ishii D, Hattori Y, Baldwin WM (2011) 3rd, Tanabe K and Fairchild RL: LFA-1 antagonism inhibits early infiltration of endogenous memory CD8 T cell into cardiac allografts and donor-reactive T cell priming. Am J Transplant 11:923-935.
Kish DD, Volokh N, Baldwin WM (2011) 3rd and Fairchild RL: Hapten application to the skin induces an inflammatory program directing hapten-primed effector CD8 T cell interaction with hapten-presenting endothelial cells. J Immunol 186: 2117-2126.
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