He has done his Ph.D., 2008, Texas A&M University, College Station, MBiot, 2003, Texas A&M, College Station, B.S., 2002, Gardner-Webb University, Boiling Springs.

The goal of our research is to identify both detrimental and repair-promoting signaling pathways resulting from neuroinflammation and glial activation then modulate them in such a way as to achieve a beneficial outcome for those suffering with neurological diseases. Members of this laboratory are investigating the physiological consequences of glial activation during infectious and autoimmune encephalitis. A major line of our current research is focused on multiple sclerosis, which I have been researching since 2003. We are particularly interested in uncovering how environmental factors, such as upper-respiratory viral infection, exacerbates symptoms of this disease as well as other neurological disease. Another emerging aspect of our research is focused on uncovering detrimental vs. repair promoting responses that occur during viral encephalitis, for which we are using the Theiler’s murine encephalomyetlitis virus model of virus-induced epilepsy to study.