Alan Nathaniel Baer

Associate Professor
Medicine - Rheumatology
Johns Hopkins Medicine
United States of America

Doctor Orthopaedics
Biography

Dr. Alan Baer graduated from the Johns Hopkins University School of Medicine in 1978 and completed his post-graduate medical training in Internal Medicine and Rheumatology at the Johns Hopkins and Vanderbilt University Hospitals. He was a faculty member at the University at Buffalo, State University of New York, from 1986 to 2007, and served there as Chief of the Section of Rheumatology and Fellowship Program Director. He joined the faculty at Johns Hopkins in 2007 and is currently Associate Professor of Medicine and Director of the Jerome L. Greene Sjogren’s Syndrome Center and the Gout Clinic. Since 2015, he been an Investigator in the Sjogren’s Syndrome Clinic at the National Institutes of Health. He was Chief of Rheumatology and Clinical Director of the Johns Hopkins University Rheumatology Practice at the Good Samaritan Hospital in Baltimore, Maryland from 2007 to 2014. Dr. Baer is currently engaged in a number of research studies in the area of Sjogren’s syndrome. He was the principal investigator of the NIH subcontract to Johns Hopkins to conduct the Sjogren’s International Registry (SICCA) and enrolled 300 patients into the registry. The SICCA registry has been a rich source of clinical data and biospecimens for research that Dr. Baer is conducting with colleagues at both Hopkins and the University of California-San Francisco. He is conducting a longitudinal observational study of patients with Sjogren’s syndrome. He is collaborating with Dr. Ben Larman in the Department of Pathology on a characterization of the complete autoantibody repertoire in patients with Sjogren’s Syndrome using phage immunoprecipation sequencing.

Research Intrest

Sjogren's Syndrome; Metabolic Myopathies

List of Publications
Baer AN, Kurano T, Thakur U, Thawait GK, Fuld MK, Maynard JW, McAdams DeMarco M, Fishman EK, Carrino JA. Sensitivity of dual-energy computed tomography for non-tophaceous and tophaceous gout.